Fig. 5From: Cohesin promotes HSV-1 lytic transcription by facilitating the binding of RNA Pol II on viral genesThe recruitment of Cohesin components by HSV-1 is independent of CTCF recruitment. BJ cells were infected with lentivirus containing control shRNA or shRNA targeting SMC1 or Rad21 for 48Â h. Then cells were infected with HSV-1 17+ for 5.5Â h and were stained with antibodies against ICP4, CTCF, SMC1, SMC3, and Rad21Back to article page