Figure 1From: The I22V and L72S substitutions in West Nile virus prM protein promote enhanced prM/E heterodimerisation and nucleocapsid incorporationGeneration and characterisation of prM22/72 mutant viruses. A) Schematics of the viruses constructed for this study, shown with or without E-glycosylation and/or prM22/72 mutations. B) Plaque morphology of recombinant viruses were performed on Baby Hamster Kidney cell monolayer, fixed and stained 4Â days post infection with crystal violet.Back to article page