Table 1 Significant pathways of the differential expressed genes in spleens of JEV-infected mice.
From: Japanese encephalitis virus infection induces changes of mRNA profile of mouse spleen and brain
Pathway name | No. of genes | p-Value |
|---|---|---|
Antigen processing and presentation | 9 | 0 |
Chemokine signaling pathway | 13 | 0 |
Complement and coagulation cascades | 7 | 0 |
Cytokine-cytokine receptor interaction | 23 | 0 |
Natural killer cell mediated cytotoxicity | 9 | 0 |
Classical Complement Pathway | 3 | 1.00E-04 |
Complement Pathway | 3 | 4.00E-04 |
Alternative Complement Pathway | 2 | 0.0021 |
Granzyme A mediated Apoptosis Pathway | 2 | 0.0036 |
Toll-like receptor signaling pathway | 4 | 0.0071 |
Intestinal immune network for IgA production | 3 | 0.009 |
CCR3 signaling in Eosinophils | 2 | 0.0111 |
Caspase Cascade in Apoptosis | 2 | 0.0121 |
NOD-like receptor signaling pathway | 3 | 0.0121 |
MAPK signaling pathway | 6 | 0.0168 |
ECM-receptor interaction | 3 | 0.0232 |
Jak-STAT signaling pathway | 4 | 0.0284 |
IFN gamma signaling pathway | 1 | 0.0478 |
B Lymphocyte Cell Surface Molecules | 1 | 0.0611 |
T cell receptor signaling pathway | 3 | 0.069 |